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TRYGENESIS
TRT & Men's Health

Fertility preservation on therapy

Exogenous testosterone suppresses the body's own production and, with it, spermatogenesis. If you intend to conceive now or later, this must be part of the conversation before therapy begins — not after.

What a provider evaluates

  • Current and future fertility intentions
  • Baseline LH, FSH and, where indicated, semen analysis
  • Whether a non-suppressive approach is more appropriate than testosterone

What treatment may involve

  • Enclomiphene, which raises endogenous testosterone without suppressing the axis
  • hCG or gonadorelin alongside testosterone in a provider-directed protocol
  • Referral for semen analysis and specialist input where appropriate

Whether any of these is appropriate is determined by a licensed provider after reviewing your history and laboratory results.

Medications a provider may consider

Questions about fertility preservation on therapy

Does testosterone therapy affect fertility?

Yes. Exogenous testosterone suppresses the hormonal signals that drive sperm production, and this can reduce or halt spermatogenesis while on therapy. If you intend to conceive now or in future, raise it before starting. Providers may consider alternatives such as enclomiphene, which raises endogenous testosterone without the same suppressive effect, or adjunct protocols alongside testosterone.

What is enclomiphene?

Enclomiphene is an oral selective estrogen receptor modulator that acts at the pituitary to increase LH and FSH, which in turn stimulate the testes to produce more testosterone. Because it works through the body's own axis rather than replacing testosterone from outside, it does not carry the same suppressive effect on sperm production. It is compounded, is not FDA-approved for this use, and is prescribed at provider discretion.